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. Study of the Synergistic Immunomodulatory and Antifibrotic Effects of Dual-Loaded Budesonide and Serpine1 siRNA Lipid–Polymer Nanoparticles Targeting Macrophage Dysregulation in Tendinopathy. ACS Applied Materials & Interfaces [Internet]. 2024;16(15):18643 - 18657. https://pubs.acs.org/doi/10.1021/acsami.4c02363
López-Cerdá et al. - 2024 - Study of the Synergistic Immunomodulatory and Antifibrotic Effects of Dual-Loaded Budesonide [...] (7.06 MB)
López-Cerdá et al. - 2024 - Study of the Synergistic Immunomodulatory and Antifibrotic Effects of Dual-Loaded Budesonide [...] (7.06 MB). Factors regulating the substrate specificity of cytosolic phospholipase A2-alpha in vitro. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 2016;1861(11):1597. Batchu et al. - 2016 - Factors regulating the substrate specificity of cy.pdf (986.25 KB)
Batchu et al. - 2016 - Factors regulating the substrate specificity of cy.pdf (986.25 KB). Lymphatic Vessels in Regenerative Medicine and Tissue Engineering. Tissue Engineering Part B [Internet]. 2016;22(5):1-13. http://online.liebertpub.com/doi/10.1089/ten.TEB.2016.0034 Schaupper et al. Tissue Engineering Part B. Review (postprint manuscript = accepted version after peer review). (697.3 KB)
Schaupper et al. Tissue Engineering Part B. Review (postprint manuscript = accepted version after peer review). (697.3 KB). Substrate efflux propensity is the key determinant of iPLA-β-mediated glycerophospholipid hydrolysised. Journal of Biological Chemistry [Internet]. 2015;. http://www.jbc.org/content/early/2015/02/23/jbc.M115.642835.abstract J. Biol. Chem.-2015-Batchu-jbc.M115.642835.pdf (786.13 KB)
J. Biol. Chem.-2015-Batchu-jbc.M115.642835.pdf (786.13 KB). CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated VEGF-C activation. Circulation [Internet]. 2014;129(19). http://circ.ahajournals.org/content/early/2014/02/19/CIRCULATIONAHA.113.002779.abstract Postprint of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated VEGF-C activation (6.6 MB) Postprint of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated VEGF-C activation (Suppl.) (2.38 MB) Published version of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated... (incl. suppl.) (23.21 MB)
Postprint of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated VEGF-C activation (6.6 MB) Postprint of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated VEGF-C activation (Suppl.) (2.38 MB) Published version of Jeltsch et al. 2014 (Circulation): CCBE1 enhances lymphangiogenesis via ADAMTS3-mediated... (incl. suppl.) (23.21 MB). The basis for the distinct biological activities of vascular endothelial growth factor receptor-1 ligands. Sci Signal. 2013;6(282):ra52. Anisimov 2013 Science Signaling (1.92 MB) Anisimov 2013 Science Signaling Supplement (2.09 MB)
Anisimov 2013 Science Signaling (1.92 MB) Anisimov 2013 Science Signaling Supplement (2.09 MB). Vascular endothelial growth factor-angiopoietin chimera with improved properties for therapeutic angiogenesis. Circulation. 2013;127(4):424-434. Anisimov 2013 (6.56 MB) Anisimov 2013 Supplement (4.93 MB)
Anisimov 2013 (6.56 MB) Anisimov 2013 Supplement (4.93 MB). Vascular endothelial growth factor-angiopoietin chimera with improved properties for therapeutic angiogenesis. Circulation. 2013;127(4):424-434. Anisimov 2013 (6.56 MB) Anisimov 2013 Supplement (4.93 MB)
Anisimov 2013 (6.56 MB) Anisimov 2013 Supplement (4.93 MB). Claudin-like protein 24 interacts with the VEGFR-2 and VEGFR-3 pathways and regulates lymphatic vessel development. Genes Dev [Internet]. 2010;24(9):875 - 80. http://view.ncbi.nlm.nih.gov/pubmed/20439428 Pipsa Saharinen et al., Genes & Development 2010 (1.2 MB) Pipsa Saharinen et al., Genes & Development 2010, supplement (9.15 MB)
Pipsa Saharinen et al., Genes & Development 2010 (1.2 MB) Pipsa Saharinen et al., Genes & Development 2010, supplement (9.15 MB). Effective suppression of vascular network formation by combination of antibodies blocking VEGFR ligand binding and receptor dimerization. Cancer Cell [Internet]. 2010;18(6):630 - 40. http://view.ncbi.nlm.nih.gov/pubmed/21130043 Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB)
Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB). Effective suppression of vascular network formation by combination of antibodies blocking VEGFR ligand binding and receptor dimerization. Cancer Cell [Internet]. 2010;18(6):630 - 40. http://view.ncbi.nlm.nih.gov/pubmed/21130043 Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB)
Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB). Effective suppression of vascular network formation by combination of antibodies blocking VEGFR ligand binding and receptor dimerization. Cancer Cell [Internet]. 2010;18(6):630 - 40. http://view.ncbi.nlm.nih.gov/pubmed/21130043 Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB)
Denis Tvorogov et al., Cancer Cell 2010 (1.3 MB) Denis Tvorogov et al., Cancer Cell 2010, supplement (828.69 KB). Suppressive effects of vascular endothelial growth factor-B on tumor growth in a mouse model of pancreatic neuroendocrine tumorigenesis. PLoS ONE [Internet]. 2010;5(11):e14109. http://view.ncbi.nlm.nih.gov/pubmed/21124841 Imke Albrecht et al., PLoS One 2010 (3.37 MB)
Imke Albrecht et al., PLoS One 2010 (3.37 MB). Vascular endothelial growth factor-B acts as a coronary growth factor in transgenic rats without inducing angiogenesis, vascular leak, or inflammation. Circulation [Internet]. 2010;122(17):1725 - 33. http://view.ncbi.nlm.nih.gov/pubmed/20937974 Maija Bry et al., Circulation 2010 (2.23 MB) Maija Bry et al., Circulation 2010, supplement (17.77 MB)
Maija Bry et al., Circulation 2010 (2.23 MB) Maija Bry et al., Circulation 2010, supplement (17.77 MB). Overexpression of vascular endothelial growth factor-B in mouse heart alters cardiac lipid metabolism and induces myocardial hypertrophy. Circ Res [Internet]. 2008;103(9):1018 - 26. http://view.ncbi.nlm.nih.gov/pubmed/18757827 Terhi Kärpänen et al., Circulation Research 2008 (898.27 KB) Terhi Kärpänen et al., Circulation Research 2008, supplement (1.56 MB)
Terhi Kärpänen et al., Circulation Research 2008 (898.27 KB) Terhi Kärpänen et al., Circulation Research 2008, supplement (1.56 MB). Overexpression of vascular endothelial growth factor-B in mouse heart alters cardiac lipid metabolism and induces myocardial hypertrophy. Circ Res [Internet]. 2008;103(9):1018 - 26. http://view.ncbi.nlm.nih.gov/pubmed/18757827 Terhi Kärpänen et al., Circulation Research 2008 (898.27 KB) Terhi Kärpänen et al., Circulation Research 2008, supplement (1.56 MB)
Terhi Kärpänen et al., Circulation Research 2008 (898.27 KB) Terhi Kärpänen et al., Circulation Research 2008, supplement (1.56 MB). The tyrosine kinase inhibitor cediranib blocks ligand-induced vascular endothelial growth factor receptor-3 activity and lymphangiogenesis. Cancer Res [Internet]. 2008;68(12):4754 - 62. http://view.ncbi.nlm.nih.gov/pubmed/18559522 Caroline Heckman et al., Cancer Research 2008 (504.8 KB)
Caroline Heckman et al., Cancer Research 2008 (504.8 KB). The tyrosine kinase inhibitor cediranib blocks ligand-induced vascular endothelial growth factor receptor-3 activity and lymphangiogenesis. Cancer Res [Internet]. 2008;68(12):4754 - 62. http://view.ncbi.nlm.nih.gov/pubmed/18559522 Caroline Heckman et al., Cancer Research 2008 (504.8 KB)
Caroline Heckman et al., Cancer Research 2008 (504.8 KB). Distinct architecture of lymphatic vessels induced by chimeric vascular endothelial growth factor-C/vascular endothelial growth factor heparin-binding domain fusion proteins. Circ Res [Internet]. 2007;100(10):1468 - 75. http://view.ncbi.nlm.nih.gov/pubmed/17478733 Tuomas Tammela et al., Circulation Research 2007 (1.1 MB)
Tuomas Tammela et al., Circulation Research 2007 (1.1 MB). Inhibiton of VEGF-C-induced VEGFR-3 activity and lymphatic endothelial cell function by the tyrosine kinase inhibitor AZD2171. In 98th Annual Meeting of the American-Association-for-Cancer-Research [Internet]. Los Angeles, CA: American Association for Cancer Research; 2007. http://dx.doi.org/10.1158/0008-5472.CAN-07-5809 Heckman et al. - 2007 - Inhibiton of VEGF-C-induced VEGFR-3 activity and l.pdf (236.37 KB)
Heckman et al. - 2007 - Inhibiton of VEGF-C-induced VEGFR-3 activity and l.pdf (236.37 KB). Inhibiton of VEGF-C-induced VEGFR-3 activity and lymphatic endothelial cell function by the tyrosine kinase inhibitor AZD2171. In 98th Annual Meeting of the American-Association-for-Cancer-Research [Internet]. Los Angeles, CA: American Association for Cancer Research; 2007. http://dx.doi.org/10.1158/0008-5472.CAN-07-5809 Heckman et al. - 2007 - Inhibiton of VEGF-C-induced VEGFR-3 activity and l.pdf (236.37 KB)
Heckman et al. - 2007 - Inhibiton of VEGF-C-induced VEGFR-3 activity and l.pdf (236.37 KB). Functional interaction of VEGF-C and VEGF-D with neuropilin receptors. FASEB J [Internet]. 2006;20(9):1462 - 72. http://view.ncbi.nlm.nih.gov/pubmed/16816121 Terhi Kärpänen et al., FASEB Journal 2006 (2.51 MB)
Terhi Kärpänen et al., FASEB Journal 2006 (2.51 MB). Pathogenesis of persistent lymphatic vessel hyperplasia in chronic airway inflammation. J Clin Invest [Internet]. 2005;115(2):247 - 57. http://view.ncbi.nlm.nih.gov/pubmed/15668734 Peter Baluk et al., Journal of Clinical Investigation 2005 (1.89 MB)
Peter Baluk et al., Journal of Clinical Investigation 2005 (1.89 MB). Vascular endothelial cell growth factor receptor 3-mediated activation of lymphatic endothelium is crucial for tumor cell entry and spread via lymphatic vessels. Cancer Res [Internet]. 2005;65(11):4739 - 46. http://view.ncbi.nlm.nih.gov/pubmed/15930292 Yulong He et al., Cancer Research 2005 (1.82 MB)
Yulong He et al., Cancer Research 2005 (1.82 MB). Vascular endothelial cell growth factor receptor 3-mediated activation of lymphatic endothelium is crucial for tumor cell entry and spread via lymphatic vessels. Cancer Res [Internet]. 2005;65(11):4739 - 46. http://view.ncbi.nlm.nih.gov/pubmed/15930292 Yulong He et al., Cancer Research 2005 (1.82 MB)
Yulong He et al., Cancer Research 2005 (1.82 MB)